Hashimoto's thyroiditis or chronic lymphocytic thyroiditis is an autoimmune disease in which the thyroid gland is slowly destroyed by a variety of cell and antibody mediated immune processes. It was the first disease to be known as an autoimmune disease. It was first described by the Japanese expert Dr. Hakaru Hashimoto in Germany in 1912. Symptoms of Hashimoto's thyroiditis include weight gain, depression, mania, sensitivity to warm and cold , paresthesia, fatigue, panic attacks, bradycardia, tachycardia, high cholesterol, hasty hypoglycemia, constipation, migraines, muscle weakness, cramps, memory loss, infertility and hair loss.
The genes concerned vary in different ethnic groups and the incidence is increased in patients with chromosomal disorders, including Turner, Down's, and Klinefelter's syndromes usually linked with autoantibodies against thyroglobulin and thyroperoxidase. The underlying specifics of the immune system destruction of thyroid cells are not clearly understood. Various autoantibodies may be present against thyroid peroxidase, thyroglobulin and TSH receptors, although a small percentage of patients may have none of these antibodies present. A percentage of the inhabitants may also have these antibodies without developing Hashimoto's thyroiditis.
The genes concerned vary in different ethnic groups and the incidence is increased in patients with chromosomal disorders, including Turner, Down's, and Klinefelter's syndromes usually linked with autoantibodies against thyroglobulin and thyroperoxidase. The underlying specifics of the immune system destruction of thyroid cells are not clearly understood. Various autoantibodies may be present against thyroid peroxidase, thyroglobulin and TSH receptors, although a small percentage of patients may have none of these antibodies present. A percentage of the inhabitants may also have these antibodies without developing Hashimoto's thyroiditis. Hypothyroidism caused by Hashimoto's Thyroiditis is treating with thyroid hormone substitute agents such as levothyroxine or desiccated thyroid extract.
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The genes concerned vary in different ethnic groups and the incidence is increased in patients with chromosomal disorders, including Turner, Down's, and Klinefelter's syndromes usually linked with autoantibodies against thyroglobulin and thyroperoxidase. The underlying specifics of the immune system destruction of thyroid cells are not clearly understood. Various autoantibodies may be present against thyroid peroxidase, thyroglobulin and TSH receptors, although a small percentage of patients may have none of these antibodies present. A percentage of the inhabitants may also have these antibodies without developing Hashimoto's thyroiditis.
The genes concerned vary in different ethnic groups and the incidence is increased in patients with chromosomal disorders, including Turner, Down's, and Klinefelter's syndromes usually linked with autoantibodies against thyroglobulin and thyroperoxidase. The underlying specifics of the immune system destruction of thyroid cells are not clearly understood. Various autoantibodies may be present against thyroid peroxidase, thyroglobulin and TSH receptors, although a small percentage of patients may have none of these antibodies present. A percentage of the inhabitants may also have these antibodies without developing Hashimoto's thyroiditis. Hypothyroidism caused by Hashimoto's Thyroiditis is treating with thyroid hormone substitute agents such as levothyroxine or desiccated thyroid extract.
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